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The important aspect to see while treating tumor patients with MET inhibitor is that tumor derived tumor necrosis factor- (TNF-) or any other inflammatory stimuli enhances the MET activation in human neutrophils and leads to their transmigration across an activated endothelium and releases free radicals, which can inhibit the cancer cell growth [76]

Inflammation-induced increases in plasma endocan levels are associated with endothelial dysfunction in humans in vivo

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Clinical Relevance: Supports mechanism of centrally-mediated satiety enhancement rather than conditioned taste aversion

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